(2019) Peroxisome Proliferator-activated Receptor (PPAR)-gamma Modifies A beta Neurotoxin-induced Electrophysiological Alterations in Rat Primary Cultured Hippocampal Neurons. Iranian Journal of Pharmaceutical Research. pp. 1403-1418. ISSN 1735-0328

(2019) Peroxisome proliferator-activated receptor (PPAR)-γ modifies Aβ neurotoxin-induced electrophysiological alterations in rat primary cultured hippocampal neurons. Iranian Journal of Pharmaceutical Research. pp. 1403-1418. ISSN 17350328 (ISSN)

(2016) Minocycline did not prevent the neurotoxic effects of amyloid beta on intrinsic electrophysiological properties of hippocampal CA1 pyramidal neurons in a rat model of Alzheimer's disease. Physiology and Pharmacology. pp. 98-107. ISSN 2476-5236

(2015) ALTERATIONS IN CA1 PYRAMIDAL NEURONAL INTRINSIC EXCITABILITY MEDIATED BY Ih CHANNEL CURRENTS IN A RAT MODEL OF AMYLOID BETA PATHOLOGY. Neuroscience. pp. 279-292. ISSN 0306-4522

(2015) Suppressive Effects of Resveratrol Treatment on The Intrinsic Evoked Excitability of CA1 Pyramidal Neurons. Cell Journal. pp. 532-539. ISSN 2228-5806

(2014) Effects of resveratrol on intrinsic neuronal properties of CA1 pyramidal neurons in rat hippocampal slices. Physiology and Pharmacology. pp. 144-155. ISSN 17350581 (ISSN)

(2013) Antibiotic supplements affect electrophysiological properties and excitability of rat hippocampal pyramidal neurons in primary culture. Iran Biomed J. pp. 101-6. ISSN 1028-852x

(2007) Forskolin potentiates the paraoxon-induced hyperexcitability in snail neurons by blocking afterhyperpolarization. Neurotoxicology. pp. 1178-1183. ISSN 0161-813X

(2007) Involvement of protein kinase C and IP3-mediated Ca2+ release in activity modulation by paraoxon in snail neurons. European Journal of Pharmacology. pp. 81-87. ISSN 0014-2999

(2006) Paraoxon suppresses Ca2+ spike and afterhyperpolarization in snail neurons: Relevance to the hyperexcitability induction. Brain Research. pp. 110-117. ISSN 0006-8993

(2006) The functional consequences of paraoxon exposure in central neurones of land snail, Caucasotachea atrolabiata, are partly mediated through modulation of Ca2+ and Ca2+-activated K+-channels. Comparative Biochemistry and Physiology C-Toxicology & Pharmacology. pp. 464-472. ISSN 1532-0456

(2004) Calcium channels as a target for paraoxon-induced neurotoxicity in snail neurons. Toxicology and Applied Pharmacology. p. 323. ISSN 0041-008X